Quantitative variation in susceptibility to Pseudomonas syringae (strain pst DC3000)


 

I have been interested in many projects related to the evolution of plant-pathogen interactions. My interest in host-pathogen interactions stems from the fact that they may help explain the relative ubiquity of recombination, outcrossing, and the maintenance of genetic diversity. However, I am also interested in mapping complex and have projects related to mapping disease and tolerance to pathogens.

The interaction between A. thaliana and P. syringae is the primary model for the study of the molecular basis of plant pathogen interactions.  Yet, we have recently found extensive quantitative variation in their interaction (Kover & Schaal 2002), which has not been previously investigated.  We have further investigated the basis of this interaction by performing QTL analysis, and diallel analysis.  We continue to do research on the genetic basis of susceptibility and characterization of tolerance to pstDC3000.

 

 

 

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University of Manchester

Division of Integrative Biology